Now, if the several lesions, cardiac and other, are due to a common cause, then why this difference in tissue response? Why do the fibroserous membranes of the joints recover unscathed, while those of the heart, also a motile organ, suffer such grievous injury? Is it that the cardiac lesions are chiefly due to streptococcal intrusion and that here, as in so many diseases, they play the part of secondary invaders? Many think so, but obviously the adjective secondary implies that the impugned streptococci, if in part, are nevertheless not wholly responsible for the cardiac lesions.
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